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tree07oxygen |

) 92 (69.2 ) 0.655 28 18 117 79 6 2 (7.1 ) 0 (.0 ) 6 (5.1 ) 5 (6.3 ) 8 (28.6 ) 10 (55.6 ) 27 (23.1 ) 14 (17.7 ) 18 (64.3 ) 8 (44.4 ) 84 (71.8 ) 60 (75.9 ) 0.161 163 18 19 11 11 5 21 6 (3.7 ) 3 (16.7 ) 0 (.0 ) 1 (9.1 ) 0 (.0 ) 1 (20.0 ) 36 (22.1 ) 6 (33.3 ) 7 (36.8 ) 1 (9.1 ) 4 (36.4 ) 1 (20.0 ) 121 (74.2 ) 9 (50.0 ) 12 (63.2 ) 9 (81.8 ) 7 (63.6 ) 3 (60.0 ) 0.077 16
) 92 (69.2 ) 0.655 28 18 117 79 6 2 (7.1 ) 0 (.0 ) 6 (5.1 ) 5 (6.3 ) 8 (28.6 ) 10 (55.6 ) 27 (23.1 ) 14 (17.7 ) 18 (64.3 ) 8 (44.4 ) 84 (71.8 ) 60 (75.9 ) 0.161 163 18 19 11 11 5 21 6 (3.7 ) 3 (16.7 ) 0 (.0 ) 1 (9.1 ) 0 (.0 ) 1 (20.0 ) 36 (22.1 ) 6 (33.3 ) 7 (36.8 ) 1 (9.1 ) 4 (36.4 ) 1 (20.0 ) 121 (74.2 ) 9 (50.0 ) 12 (63.2 ) 9 (81.8 ) 7 (63.6 ) 3 (60.0 ) 0.077 16
) 92 (69.2 ) 0.655 28 18 117 79 6 2 (7.1 ) 0 (.0 ) 6 (5.1 ) 5 (6.3 ) 8 (28.6 ) 10 (55.6 ) 27 (23.1 ) 14 (17.7 ) 18 (64.3 ) 8 (44.4 ) 84 (71.8 ) 60 (75.9 ) 0.161 163 18 19 11 11 5 21 6 (3.7 ) 3 (16.7 ) 0 (.0 ) 1 (9.1 ) 0 (.0 ) 1 (20.0 ) 36 (22.1 ) 6 (33.3 ) 7 (36.8 ) 1 (9.1 ) 4 (36.4 ) 1 (20.0 ) 121 (74.2 ) 9 (50.0 ) 12 (63.2 ) 9 (81.8 ) 7 (63.6 ) 3 (60.0 ) 0.077 16
Is still the most lethal gynecologic malignancy worldwide. Although most ovarian carcinoma cells are initially sensitive to chemotherapy, there is always a small population cells that always survives and initiates new tumors which causes recurrence [31]. The verification of tumor heterogeneity further enhances the hypothesis of CSCs. Compelling evidence has shown that ovarian carcinomas with enri
Is still the most lethal gynecologic malignancy worldwide. Although most ovarian carcinoma cells are initially sensitive to chemotherapy, there is always a small population cells that always survives and initiates new tumors which causes recurrence [31]. The verification of tumor heterogeneity further enhances the hypothesis of CSCs. Compelling evidence has shown that ovarian carcinomas with enri
Is still the most lethal gynecologic malignancy worldwide. Although most ovarian carcinoma cells are initially sensitive to chemotherapy, there is always a small population cells that always survives and initiates new tumors which causes recurrence [31]. The verification of tumor heterogeneity further enhances the hypothesis of CSCs. Compelling evidence has shown that ovarian carcinomas with enri
Etc. [34, 35]. However, it still remains challenging to identify one single marker or several combined markers to specifically identify all the CSCs in ovarian tumor, and the exact roles of these `stemness' related markers, are still poorly understood due to either a current lack of understanding of the biological functions of the markers, or frequently the lack of information correlating the var
Etc. [34, 35]. However, it still remains challenging to identify one single marker or several combined markers to specifically identify all the CSCs in ovarian tumor, and the exact roles of these `stemness' related markers, are still poorly understood due to either a current lack of understanding of the biological functions of the markers, or frequently the lack of information correlating the var